by chris c Mon May 15 2017, 23:02
Seen Dave Feldman's experiments?
http://cholesterolcode.comA lot of interesting and seemingly non-logical stuff. Graham you appear to be on the opposite extreme with your HDL. I'd say your LDL was pretty much where you need it to be at your age (like me).
Originally it looked like my LDL went up on low carb, and then came back down when I added more saturated fats. In retrospect it may have gone up while I was losing the dietician - added weight, and back down again when I reverted to my original weight, not uncommon when body fat is being liberated into the circulation.
The complete and utter weirdness is that when my thyroid blew up the LDL came down by exactly the same amount as the statin reduced it. When the thyroid was overtreated it shot up again. Yet it was when the thyroid was high and the "cholesterol" low that my arteries clagged up, and when the thyroid came down and my LDL went back up again that they unclogged. So another epic fail in the cholesterol causes CVD argument.
It looks to me as if high trigs are a marker for carb consumption (especially fructose) in the absence of sufficient fat, and HDL is a marker for fat consumption in the absence of excess carbs. So no surprise that trigs/HDL is a marker for insulin resistance/high insulin, and that is more likely to do the damage that is blamed on the LDL. High glucose and high insulin both damage the endothelium through different pathways, and they also cause malformed (small dense) LDL and damage it through oxidation and glycation, so when LDL arrives to heal the damage it doesn't work properly.
I visualise firemen turning up drunk on duty (sorry, Paul!) and ending up in a pile on the floor getting in the way of the sober ones. But they didn't start the fire.